JCI table of contents: January 2, 2007

In an accompanying commentary, Siamon Gordon from the University of Oxford discusses how these three studies yield further insights into the complexity of monocytes and tissue macrophages during genetic- and high-fat diet–induced disease. He concludes that further investigation is required before different subsets of cells can be selectively, efficiently, and safely targeted for potential therapies for conditions such as atherosclerosis or insulin resistance.

TITLE: Ly-6Chi monocytes dominate hypercholesterolemia-associated monocytosis and give rise to macrophages in atheromata

AUTHOR CONTACT:

Mikael Pittet
Massachusetts General Hospital, Boston, Massachusetts, USA.
Phone: (617) 726-5788; Fax: (617) 726-5708; E-mail: .
View the PDF of this article at: https://www.the-jci.org/article.php?id=29950

RELATED MANUSCRIPT

TITLE: Monocyte subsets differentially employ CCR2, CCR5, and CX3CR1 to accumulate within atherosclerotic plaques

AUTHOR CONTACT:

Gwendalyn Randolph
Mount Sinai School of Medicine, New York, New York, USA.
Phone: (212) 659-8262; Fax: (212) 803-6740; E-mail: .

View the PDF of this article at: https://www.the-jci.org/article.php?id=28549

RELATED MANUSCRIPT

TITLE: Obesity induces a phenotypic switch in adipose tissue macrophage polarization

AUTHOR CONTACT:

Alan Saltiel
University of Michigan, Ann Arbor, Michigan, USA.
Phone: (734) 615-9787; Fax: (734) 763-6492; E-mail: .

View the PDF of this article at:https://www.the-jci.org/article.php?id=29881

ACCOMPANYING COMMENTARY

TITLE: Macrophage heterogeneity and tissue lipids

AUTHOR CONTACT:

Siamon Gordon
University of Oxford, Oxford, United Kingdom.
Phone: +44-1865-275534; Fax: +44-1865-275515; E-mail: or .
View the PDF of this article at: https://www.the-jci.org/article.php?id=30992